thp 1 aml Search Results


human monocytic aml cells  (ATCC)
99
ATCC human monocytic aml cells
Human Monocytic Aml Cells, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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aml cell lines  (Procell Inc)
86
Procell Inc aml cell lines
MEF2A expression was upregulated in <t>AML.</t> ( A ) Analysis of MEF2A expression across pan-cancer samples. ( B ) The GEPIA database was used to analyze MEF2A expression in AML tissues and normal samples. ( C ) MEF2A mRNA expression was detected by qRT-PCR in AML samples ( N = 42) and healthy samples ( N = 16). ( D ) The western blotting assay was performed to detect MEF2A protein expression in AML samples and healthy samples. ( E ) MEF2A mRNA expression was detected by qRT-PCR in HS-5, <t>HL-60,</t> <t>MOLM-13,</t> THP-1, <t>U937,</t> and KG-1 cells. ( F ) MEF2A protein expression was analyzed by western blotting assay in HS-5 cells, HL-60 cells, and KG-1 cells
Aml Cell Lines, supplied by Procell Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/aml+cell+lines/pmc13141345-96-0-7
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human aml cell lines  (ATCC)
99
ATCC human aml cell lines
MEF2A expression was upregulated in <t>AML.</t> ( A ) Analysis of MEF2A expression across pan-cancer samples. ( B ) The GEPIA database was used to analyze MEF2A expression in AML tissues and normal samples. ( C ) MEF2A mRNA expression was detected by qRT-PCR in AML samples ( N = 42) and healthy samples ( N = 16). ( D ) The western blotting assay was performed to detect MEF2A protein expression in AML samples and healthy samples. ( E ) MEF2A mRNA expression was detected by qRT-PCR in HS-5, <t>HL-60,</t> <t>MOLM-13,</t> THP-1, <t>U937,</t> and KG-1 cells. ( F ) MEF2A protein expression was analyzed by western blotting assay in HS-5 cells, HL-60 cells, and KG-1 cells
Human Aml Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/HL-60/10__1158_slash_2159___8290__cd___17___0399-268-25-37
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human aml cell lines - by Bioz Stars, 2026-09
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aml cell lines thp 1  (DSMZ)
96
DSMZ aml cell lines thp 1
Figure 1: )e inhibitory effect of HHTon nine <t>AML</t> <t>cell</t> lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.
Aml Cell Lines Thp 1, supplied by DSMZ, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/THP-1/pm36081671-35-0-39
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aml cell lines  (ATCC)
96
ATCC aml cell lines
Figure 1: )e inhibitory effect of HHTon nine <t>AML</t> <t>cell</t> lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.
Aml Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/C1498/pm40664450-47-1-30
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aml cell lines - by Bioz Stars, 2026-09
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thp-1  (CEM Corporation)
90
CEM Corporation thp-1
Figure 1: )e inhibitory effect of HHTon nine <t>AML</t> <t>cell</t> lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.
Thp 1, supplied by CEM Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/ccrf+cem+cells/pm36120594-116-4-10
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human monocytic aml cell lines  (ATCC)
99
ATCC human monocytic aml cell lines
Figure 1: )e inhibitory effect of HHTon nine <t>AML</t> <t>cell</t> lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.
Human Monocytic Aml Cell Lines, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/MV-4-11/pmc09534319-41-0-12
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thp 1 aml atcc  (ATCC)
93
ATCC thp 1 aml atcc
Figure 1: )e inhibitory effect of HHTon nine <t>AML</t> <t>cell</t> lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.
Thp 1 Aml Atcc, supplied by ATCC, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/thp+1+aml/Cellulomonas+biazotea/us10751422-1883-84-86
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aml cell lines  (DSMZ)
96
DSMZ aml cell lines
Ligand profile characterization of leukemia cell lines. Percentage of ligand or GFP-expressing cells was determined by flow cytometry. ( a ) Ligand expression in B-ALL cell lines (KOPN-8, MHH-CALL-4, Nalm-6 and Nalm-6 GFP/Luc). ( b ) GFP expression of Nalm-6 GFP/Luc (grey) in comparison to non-transduced Nalm-6 (white). ( c ) Ligand expression in <t>AML</t> <t>cell</t> lines (NOMO-1, THP-1, U-937 and U-937 CD19tag/Luc). ( d ) CD19 expression of U-937 CD19tag/Luc (grey) in comparison to non-transduced U-937 (white).
Aml Cell Lines, supplied by DSMZ, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


MEF2A expression was upregulated in AML. ( A ) Analysis of MEF2A expression across pan-cancer samples. ( B ) The GEPIA database was used to analyze MEF2A expression in AML tissues and normal samples. ( C ) MEF2A mRNA expression was detected by qRT-PCR in AML samples ( N = 42) and healthy samples ( N = 16). ( D ) The western blotting assay was performed to detect MEF2A protein expression in AML samples and healthy samples. ( E ) MEF2A mRNA expression was detected by qRT-PCR in HS-5, HL-60, MOLM-13, THP-1, U937, and KG-1 cells. ( F ) MEF2A protein expression was analyzed by western blotting assay in HS-5 cells, HL-60 cells, and KG-1 cells

Journal: Biology Direct

Article Title: MEF2A activates the ZDHHC20/NF-κB pathway to inhibit doxorubicin sensitivity and promote the malignant progression of acute myeloid leukemia

doi: 10.1186/s13062-026-00762-y

Figure Lengend Snippet: MEF2A expression was upregulated in AML. ( A ) Analysis of MEF2A expression across pan-cancer samples. ( B ) The GEPIA database was used to analyze MEF2A expression in AML tissues and normal samples. ( C ) MEF2A mRNA expression was detected by qRT-PCR in AML samples ( N = 42) and healthy samples ( N = 16). ( D ) The western blotting assay was performed to detect MEF2A protein expression in AML samples and healthy samples. ( E ) MEF2A mRNA expression was detected by qRT-PCR in HS-5, HL-60, MOLM-13, THP-1, U937, and KG-1 cells. ( F ) MEF2A protein expression was analyzed by western blotting assay in HS-5 cells, HL-60 cells, and KG-1 cells

Article Snippet: AML cell lines (MOLM-13, THP-1, and U937, Procell, Wuhan, China) were cultured in RPMI-1640 added with 10% fetal bovine serum (EK-Bioscience) and 1% penicillin/streptomycin (Millipore) at 37°C with 5% CO 2 .

Techniques: Expressing, Quantitative RT-PCR, Western Blot

Figure 1: )e inhibitory effect of HHTon nine AML cell lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.

Journal: Journal of oncology

Article Title: Characterization of the Newly Established Homoharringtonine- (HHT-) Resistant Cell Lines and Mechanisms of Resistance.

doi: 10.1155/2022/2813938

Figure Lengend Snippet: Figure 1: )e inhibitory effect of HHTon nine AML cell lines. (a) )e IC50 values of HHTon nine AML cell lines. (b) )e inhibitory effect of HHT on FLT3-ITD mutant cell lines was significantly higher than that in nonmutant ones. ∗P < 0.05.

Article Snippet: AML cell lines THP-1 (CVCL_0006), HL-60 (CVCL_A794), NB4 (CVCL-0005), Kasumi-1 (CVCL_0589), MOLM-13 (CVCL_2119), MV4-11 (CVCL_0064), KG-1 (CVCL_0374), and U937 (CVCL_0007) were purchased from American Type Culture Collection (ATCC), and OCI-AML3 (CVCL_1844) was purchased from Deutsche Sammlung vonMikroorganismen und Zellkulturen (DSMZ).

Techniques: Mutagenesis

Figure 6: Verification of the hub genes (CALCRL and GNAI1) and the prognostic significance of CALCRL and GNAI1 for AML. (a) )e mRNA level of GPR183, CNR2, CALCRL, and GNAI1 in four AML cell lines by RT-qPCR. (b) )e protein level of CALCRL and GNAI1 in four AML cell lines and the mice tumor mass from HHT-sensitive and HHT-resistant mice groups. (c) )e expression level and OS of GNAI1 and CALCRL in online database GEPIA in AML. (d) )e OS and EFS of CALCRL in our cohorts. (e) )e correlation of GNAI1 and CALCRL through correlation analysis. P < 0.01∗∗and P < 0.001∗∗∗.

Journal: Journal of oncology

Article Title: Characterization of the Newly Established Homoharringtonine- (HHT-) Resistant Cell Lines and Mechanisms of Resistance.

doi: 10.1155/2022/2813938

Figure Lengend Snippet: Figure 6: Verification of the hub genes (CALCRL and GNAI1) and the prognostic significance of CALCRL and GNAI1 for AML. (a) )e mRNA level of GPR183, CNR2, CALCRL, and GNAI1 in four AML cell lines by RT-qPCR. (b) )e protein level of CALCRL and GNAI1 in four AML cell lines and the mice tumor mass from HHT-sensitive and HHT-resistant mice groups. (c) )e expression level and OS of GNAI1 and CALCRL in online database GEPIA in AML. (d) )e OS and EFS of CALCRL in our cohorts. (e) )e correlation of GNAI1 and CALCRL through correlation analysis. P < 0.01∗∗and P < 0.001∗∗∗.

Article Snippet: AML cell lines THP-1 (CVCL_0006), HL-60 (CVCL_A794), NB4 (CVCL-0005), Kasumi-1 (CVCL_0589), MOLM-13 (CVCL_2119), MV4-11 (CVCL_0064), KG-1 (CVCL_0374), and U937 (CVCL_0007) were purchased from American Type Culture Collection (ATCC), and OCI-AML3 (CVCL_1844) was purchased from Deutsche Sammlung vonMikroorganismen und Zellkulturen (DSMZ).

Techniques: Quantitative RT-PCR, Expressing

Ligand profile characterization of leukemia cell lines. Percentage of ligand or GFP-expressing cells was determined by flow cytometry. ( a ) Ligand expression in B-ALL cell lines (KOPN-8, MHH-CALL-4, Nalm-6 and Nalm-6 GFP/Luc). ( b ) GFP expression of Nalm-6 GFP/Luc (grey) in comparison to non-transduced Nalm-6 (white). ( c ) Ligand expression in AML cell lines (NOMO-1, THP-1, U-937 and U-937 CD19tag/Luc). ( d ) CD19 expression of U-937 CD19tag/Luc (grey) in comparison to non-transduced U-937 (white).

Journal: International Journal of Molecular Sciences

Article Title: Preclinical Evaluation of CRISPR-Edited CAR-NK-92 Cells for Off-the-Shelf Treatment of AML and B-ALL

doi: 10.3390/ijms232112828

Figure Lengend Snippet: Ligand profile characterization of leukemia cell lines. Percentage of ligand or GFP-expressing cells was determined by flow cytometry. ( a ) Ligand expression in B-ALL cell lines (KOPN-8, MHH-CALL-4, Nalm-6 and Nalm-6 GFP/Luc). ( b ) GFP expression of Nalm-6 GFP/Luc (grey) in comparison to non-transduced Nalm-6 (white). ( c ) Ligand expression in AML cell lines (NOMO-1, THP-1, U-937 and U-937 CD19tag/Luc). ( d ) CD19 expression of U-937 CD19tag/Luc (grey) in comparison to non-transduced U-937 (white).

Article Snippet: AML cell lines (NOMO-1, THP-1, U-937) and B-ALL cell lines (KOPN-8, MHH-CALL-4, Nalm-6) were obtained from the German Collection of Microorganisms and Cell Cultures GmbH (DSMZ, Braunschweig, Germany).

Techniques: Expressing, Flow Cytometry, Comparison

Cytotoxicity assays in AML cell lines comparing parental NK-92 and CD276-CAR-NK-92 vs. irradiated, CBLB, NKG2A, TIGIT or triple knock-out cells. Specific lysis is shown as mean ± SD ( n = 3). Calcein assays were incubated for 2 h and several effector-to-target (E:T) ratios were employed: 10:1, 5:1, 2.5:1. Luciferase assays were studied for a time span of 6 h at 5:1 E:T ratio. ( a ) NK-92 cell lines vs. NOMO-1 calcein assay. ( b ) CD276-CAR-NK-92 cell lines vs. NOMO-1 calcein assay. ( c ) NK-92 cell lines vs. THP-1 calcein assay. ( d ) CD276-CAR-NK-92 cell lines vs. THP-1 calcein assay. ( e ) NK-92 cell lines vs. U-937 calcein assay. ( f ) CD276-CAR-NK-92 cell lines vs. U-937 calcein assay. ( g ) NK-92 cell lines vs. U-937 CD19tag/Luc luciferase assay. ( h ) CD276-CAR-NK-92 cell lines vs. U-937 CD19tag/Luc luciferase assay. * p < 0.05; ** p < 0.01; *** p > 0.001; **** p < 0.0001; ns, non-significant ( p > 0.05).

Journal: International Journal of Molecular Sciences

Article Title: Preclinical Evaluation of CRISPR-Edited CAR-NK-92 Cells for Off-the-Shelf Treatment of AML and B-ALL

doi: 10.3390/ijms232112828

Figure Lengend Snippet: Cytotoxicity assays in AML cell lines comparing parental NK-92 and CD276-CAR-NK-92 vs. irradiated, CBLB, NKG2A, TIGIT or triple knock-out cells. Specific lysis is shown as mean ± SD ( n = 3). Calcein assays were incubated for 2 h and several effector-to-target (E:T) ratios were employed: 10:1, 5:1, 2.5:1. Luciferase assays were studied for a time span of 6 h at 5:1 E:T ratio. ( a ) NK-92 cell lines vs. NOMO-1 calcein assay. ( b ) CD276-CAR-NK-92 cell lines vs. NOMO-1 calcein assay. ( c ) NK-92 cell lines vs. THP-1 calcein assay. ( d ) CD276-CAR-NK-92 cell lines vs. THP-1 calcein assay. ( e ) NK-92 cell lines vs. U-937 calcein assay. ( f ) CD276-CAR-NK-92 cell lines vs. U-937 calcein assay. ( g ) NK-92 cell lines vs. U-937 CD19tag/Luc luciferase assay. ( h ) CD276-CAR-NK-92 cell lines vs. U-937 CD19tag/Luc luciferase assay. * p < 0.05; ** p < 0.01; *** p > 0.001; **** p < 0.0001; ns, non-significant ( p > 0.05).

Article Snippet: AML cell lines (NOMO-1, THP-1, U-937) and B-ALL cell lines (KOPN-8, MHH-CALL-4, Nalm-6) were obtained from the German Collection of Microorganisms and Cell Cultures GmbH (DSMZ, Braunschweig, Germany).

Techniques: Irradiation, Knock-Out, Lysis, Incubation, Luciferase